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@ARTICLE{Fournier:186296,
author = {Fournier, Claudia and Winter, Marcus and Zahnreich,
Sebastian and Nasonova, Elena and Melnikova, Larissa and
Ritter, Sylvia},
title = {{I}nterrelation amongst differentiation, senescence and
genetic instability in long-term cultures of fibroblasts
exposed to different radiation qualities.},
journal = {Radiotherapy and oncology},
volume = {83},
number = {3},
issn = {0167-8140},
address = {Amsterdam [u.a.]},
publisher = {Elsevier Science},
reportid = {GSI-2016-00794},
pages = {277 - 282},
year = {2007},
abstract = {The goal of the present study was to investigate aging and
genetic instability in the progeny of human fibroblasts
exposed to X-rays and carbon ions.Following irradiation,
cells were regularly subcultured until senescence. At
selected time-points BrdU-labelling index, expression of
cell cycle related proteins, cell differentiation pattern
and chromosome aberrations were assessed.After exposure, an
immediate cell cycle arrest occurred followed by a period of
a few weeks where premature differentiation and senescence
were observed. In all cultures cycling cells expressing low
levels of cell cycle inhibiting proteins were present and
finally dominated the populations. About 5months after
exposure, the cellular and molecular changes attributed to
differentiation and senescence reappeared and persisted.
Concurrently, genetic instability was observed, but the
aberration yields and types differed between repeated
experiments. The descendants of cells exposed to carbon ions
did not senesce earlier and displayed a similar rate of
genetic instability as the X-ray progeny. For high doses an
impaired cell cycle regulation and extended life span was
observed, but finally cell proliferation ceased in all
populations.The descendants of irradiated fibroblasts
undergo stepwise senescence and differentiation. Genetic
instability is frequent and an extension of the life span
may occur.},
keywords = {Cell Cycle Proteins (NLM Chemicals) / Ions (NLM Chemicals)
/ Carbon (NLM Chemicals)},
cin = {BIO},
ddc = {610},
cid = {I:(DE-Ds200)BIO-20160831OR354},
pnm = {899 - ohne Topic (POF3-899) / 310 - Krebsforschung
(POF1-300)},
pid = {G:(DE-HGF)POF3-899 / G:(DE-HGF)POF1-310},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:17499869},
UT = {WOS:000248070300009},
doi = {10.1016/j.radonc.2007.04.022},
url = {https://repository.gsi.de/record/186296},
}